| dc.description.abstract | The "riddle of heredity" which stumped Charles Darwin and which was partially solved by Gregor Mendel in 1865, is still a riddle to today’s "enlightened" generation. As we approach the centennial of Mendel's remarkable discoveries, we find there is still much to be known about those elusive substances called "genes" and the still more nebulous matter of how they affect the phenotype. Much information can be gained on this natter by the study of mutations. Besides studying the phenomena for their own sakes, we can often determine the nature of the normal pattern of events by studying the abnormal.
The purpose of this paper, then, is to study the phenotypic effects of artificial mutation, particularly in bacteria, both by direct observation and by studying the results of others’ direct observation.The "riddle of heredity" which stumped Charles Darwin and which was partially solved by Gregor Mendel in 1865, is still a riddle to today’s "enlightened" generation. As we approach the centennial of Mendel's remarkable discoveries, we find there is still much to be known about those elusive substances called "genes" and the still more nebulous matter of how they affect the phenotype. Much information can be gained on this natter by the study of mutations. Besides studying the phenomena for their own sakes, we can often determine the nature of the normal pattern of events by studying the abnormal.
The purpose of this paper, then, is to study the phenotypic effects of artificial mutation, particularly in bacteria, both by direct observation and by studying the results of others’ direct observation.The "riddle of heredity" which stumped Charles Darwin and which was partially solved by Gregor Mendel in 1865, is still a riddle to today’s "enlightened" generation. As we approach the centennial of Mendel's remarkable discoveries, we find there is still much to be known about those elusive substances called "genes" and the still more nebulous matter of how they affect the phenotype. Much information can be gained on this natter by the study of mutations. Besides studying the phenomena for their own sakes, we can often determine the nature of the normal pattern of events by studying the abnormal.
The purpose of this paper, then, is to study the phenotypic effects of artificial mutation, particularly in bacteria, both by direct observation and by studying the results of others’ direct observation.The "riddle of heredity" which stumped Charles Darwin and which was partially solved by Gregor Mendel in 1865, is still a riddle to today’s "enlightened" generation. As we approach the centennial of Mendel's remarkable discoveries, we find there is still much to be known about those elusive substances called "genes" and the still more nebulous matter of how they affect the phenotype. Much information can be gained on this natter by the study of mutations. Besides studying the phenomena for their own sakes, we can often determine the nature of the normal pattern of events by studying the abnormal.
The purpose of this paper, then, is to study the phenotypic effects of artificial mutation, particularly in bacteria, both by direct observation and by studying the results of others’ direct observation.The "riddle of heredity" which stumped Charles Darwin and which was partially solved by Gregor Mendel in 1865, is still a riddle to today’s "enlightened" generation. As we approach the centennial of Mendel's remarkable discoveries, we find there is still much to be known about those elusive substances called "genes" and the still more nebulous matter of how they affect the phenotype. Much information can be gained on this natter by the study of mutations. Besides studying the phenomena for their own sakes, we can often determine the nature of the normal pattern of events by studying the abnormal.
The purpose of this paper, then, is to study the phenotypic effects of artificial mutation, particularly in bacteria, both by direct observation and by studying the results of others’ direct observation. | |
